SID Annual Meeting
2020-05-13 07:00:00
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  • Wed., May 13, 2020
  • Thursday, May 14, 2020
  • Friday, May 15, 2020
  • Saturday, May 16, 2020
  • Home
  • Information
    • Associate Groups
    • 2020 Meeting FAQ’s
    • Program Book
    • Abstracts
    • CME
    • Donate
    • Rothman Award
  • Wed., May 13, 2020
  • Thursday, May 14, 2020
  • Friday, May 15, 2020
  • Saturday, May 16, 2020

Plenary Session II Agenda

Presiders: Kurt Lu, MD and Peggy Myung, MD/PhD 

 

3:30 PM-3:45 PM 

#561,  Development and first-in-human characterization of a potent oral CCR4 antagonist for the treatment of atopic dermatitis 

  1. L.E. Cheng, A.Jorapur, S. Jacobson, O.Talay, S. Miakicheva, D. Trujillo, N. Lee, J. Jankicevic, D. Wustrow, P. Kassner, W. Ho, D. Brockstedt 

RAPT Therapeutics, Inc., South San Francisco, California, United States 

 

3:45 PM -4:00 PM 

#327 , Staphylococcus epidermidis protease EcpA is a deleterious component of the skin microbiome in atopic dermatitis 

  1. L. Cau1, 2, M. Williams1, A. Butcher1, T. Nakatsuji1, J. Cheng1, T. Hata1, J. Kavanaugh3, C. Mainzer2, B. Closs2, A. Horswill3, R. L. Gallo1

1Dermatology Department, University of California, San Diego, California, United States, 2R&D Department, SILAB, Brive, France, 3Immunology and Microbiology Department, University of Colorado Anschutz Medical Campus, Aurora, Colorado, United States 

 

4:00 PM-4:15 PM 

#717,  Enhanced molecular signatures in cutaneous lupus erythematosus patients support distinct pathogenic pathways in African American patients 

  1. J. L. Zhu1, L. Tran2, F. Zheng2, J. James2, J. Guthridge2, B. F. Chong1

1Dermatology, University of Texas Southwestern Medical Center, Dallas, Texas, United States, 2Arthritis & Clinical Immunology, Oklahoma Medical Research Foundation, Oklahoma City, Oklahoma, United States 

 

4:15 PM-4:30 PM 

#775,  Hyperactivation of sympathetic nerves drives melanocyte stem cell depletion 

  1. B. Zhang1, S. Ma1, 2, 3, I. Rachman4, M. He1, 5, P. Baral6, S. Choi1, W. A. Gonçalves10, Y. Shwartz1, E. M. Fast1, 7,   Y. Hsu4, L. I. Zon1, 7, 8, A. Regev2, 8, 3, J. D. Buenrostro1, T. M. Cunha6, 9, I. M. Chiu6, D. Fisher4, Y. Hsu1

1Stem Cell and Regenerative Biology, Harvard University, Cambridge, Massachusetts, United States, 2Klarman Cell Observatory, Broad Institute of MIT and Harvard, Cambridge, Massachusetts, United States, 3Department of Biology and Koch Institute, Massachusetts Institute of Technology, Cambridge, Massachusetts, United States, 4Cutaneous Biology Research Center, Department of Dermatology, Massachusetts General Hospital, Charlestown, Massachusetts, United States, 5Molecular and Cellular Biology, Harvard University, Cambridge, Massachusetts, United States, 6Department of Immunology, Harvard Medical School, Boston, Massachusetts, United States, 7Division of Hematology/Oncology, Boston Children’s Hospital/Dana-Farber Cancer Institute, Boston, Massachusetts, United States, 8Howard Hughes Medical Institute, Chevy Chase, Maryland, United States, 9Department of Pharmacology, Ribeirão Preto Medical School, University of São Paulo, Ribeirão Preto, Brazil, 10Graduate Program in Cellular Biology, Institute of Biological Science, Federal University of Minas Gerais, Belo Horizonte, Brazil 

 

4:30 PM-4:45 PM 

#030,  VGLL3, an orchestrator of female-biased autoimmunity, interfaces with the Hippo pathway to modulate genes involved in immunity and fibrosis 

  1. A.C. Billi1, C. Zeng1, M. Gharaee-Kermani2, S. W. Stoll1, M. J. Wilson1, O. Plazyo1, X. Xing1, J. M. McCarthy1, L. C. Tsoi4, 5, J. Kahlenberg2, A. A. Dlugosz1, 3, J. E. Gudjonsson1

1Dermatology, University of Michigan, Ann Arbor, Michigan, United States, 2Internal Medicine, Division of Rheumatology, University of Michigan, Ann Arbor, Michigan, United States, 3Cell and Developmental Biology, University of Michigan, Ann Arbor, Michigan, United States, 4Biostatistics, University of Michigan, Ann Arbor, Michigan, United States, 5Computational Medicine & Bioinformatics, University of Michigan, Ann Arbor, Michigan, United States 

Other Speakers

2019-10-16_1107_DiazLuis Diaz, MD
2019-04-12 Werth Portraits-64Victoria Werth, MD
Lumpkin, EllenEllen Lumpkin, PhD
HeadshotWilson Liao, MD
Garza headshot 2019Luis Garza, MD/PhD
Dr. Valentina GrecoValentina Greco, PhD
Nunez4x6Gabriel Nunez, MD
5x5GrinnellFrederick Grinnell, PhD
Bulletin Spring 2005: Zhijian Chen, University of Texas Southwestern Medical Center at Dallas - Dallas, TX / Bulletin Sep 2005: Researchers have discovered a surprise lurking inside mitochondria, the power plants that are present in every cell.  It turns out that these powerhouses also contain a protein that triggers the immune system to attack viral invaders.  According to the researchers, the new role makes perfect biological and evolutionary sense because it fits well with another function of mitochondria as executioners of a biochemical cascade that causes programmed cell death, or apoptosis.  "This is the first protein known to be involved in the immune response that is found in mitochondria," said Zhijian "James" Chen, an HHMI investigator at the University of Texas Southwestern Medical Center.  Chen and his colleagues reported the discovery on August 25, 2005, in an immediate early publication of the journal Cell.Zhijian (James) Chen, PhD
AlicePPentland,MD (2)Alice Pentland, MD
Valatine, HannahHannah Valantine, MD
Ezhkova PictureElena Ezhkova, PhD
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